Monografie
Functional analysis of ion channels and neuronal networks in 2D and 3D \(in\) \(vitro\) cell culture models
- Sprache
-
Englisch
- Identifier
-
1249073529
Thema
Cell culture; Ion channels; GABA; Functional analysis; 3D-Zellkultur; Nervennetz; Ionenkanal; Glycinrezeptor; GABA-Rezeptor
Beteiligte Personen und Organisationen
Janzen, Dieter
Villmann, Carmen
Dalton, Paul
Strissel, Pamela
Villmann, Carmen
Dalton, Paul
Strissel, Pamela
- DOI
-
10.25972/OPUS-25170
- URN
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
26.01.2023, 13:53 MEZ
Objekttyp
- Monografie
Beteiligte
- Janzen, Dieter
- Villmann, Carmen
- Dalton, Paul
- Strissel, Pamela
Ähnliche Objekte (12)
Cortical Neurons form a Functional Neuronal Network in a 3D Printed Reinforced Matrix
3D Electrophysiological Measurements on Cells Embedded within Fiber-Reinforced Matrigel
Brain and Breast Cancer Cells with PTEN Loss of Function Reveal Enhanced Durotaxis and RHOB Dependent Amoeboid Migration Utilizing 3D Scaffolds and Aligned Microfiber Tracts
Methods to induce polarity in constructed neuronal networks
Reinforced Hyaluronic Acid-Based Matrices Promote 3D Neuronal Network Formation
Hybrid hydrogels promote physiological functionality of long-term cultured primary neuronal cells in vitro
Novel neuro-microphysiological system (nMPS) for multiple characterisations of 3D neuronal networks co-cultured with astrocytes and microglia
A novel role of B-RAF in the regulation of ion channels and transporters
Beyond standard assumptions on neural excitability
Expression von Kv1.3-Kaliumkanälen in retinalen Pigmentepithelzellen
Die Mutation Y1206S steigert die Affinität des Sulfonylharnstoffrezeptors SUR2A für Glibenclamid und unterstützt den Einfluß der Koexpression mit KIR6.2
Genetic programming of the visual forebrain in the absence of retinal input
Cortical Neurons form a Functional Neuronal Network in a 3D Printed Reinforced Matrix
3D Electrophysiological Measurements on Cells Embedded within Fiber-Reinforced Matrigel
Brain and Breast Cancer Cells with PTEN Loss of Function Reveal Enhanced Durotaxis and RHOB Dependent Amoeboid Migration Utilizing 3D Scaffolds and Aligned Microfiber Tracts
Methods to induce polarity in constructed neuronal networks
Reinforced Hyaluronic Acid-Based Matrices Promote 3D Neuronal Network Formation
Hybrid hydrogels promote physiological functionality of long-term cultured primary neuronal cells in vitro
Novel neuro-microphysiological system (nMPS) for multiple characterisations of 3D neuronal networks co-cultured with astrocytes and microglia
A novel role of B-RAF in the regulation of ion channels and transporters
Beyond standard assumptions on neural excitability
Expression von Kv1.3-Kaliumkanälen in retinalen Pigmentepithelzellen
Die Mutation Y1206S steigert die Affinität des Sulfonylharnstoffrezeptors SUR2A für Glibenclamid und unterstützt den Einfluß der Koexpression mit KIR6.2
Genetic programming of the visual forebrain in the absence of retinal input
Cortical Neurons form a Functional Neuronal Network in a 3D Printed Reinforced Matrix
3D Electrophysiological Measurements on Cells Embedded within Fiber-Reinforced Matrigel
Brain and Breast Cancer Cells with PTEN Loss of Function Reveal Enhanced Durotaxis and RHOB Dependent Amoeboid Migration Utilizing 3D Scaffolds and Aligned Microfiber Tracts
Methods to induce polarity in constructed neuronal networks
Reinforced Hyaluronic Acid-Based Matrices Promote 3D Neuronal Network Formation
Hybrid hydrogels promote physiological functionality of long-term cultured primary neuronal cells in vitro
Novel neuro-microphysiological system (nMPS) for multiple characterisations of 3D neuronal networks co-cultured with astrocytes and microglia
A novel role of B-RAF in the regulation of ion channels and transporters
Beyond standard assumptions on neural excitability
Expression von Kv1.3-Kaliumkanälen in retinalen Pigmentepithelzellen
Die Mutation Y1206S steigert die Affinität des Sulfonylharnstoffrezeptors SUR2A für Glibenclamid und unterstützt den Einfluß der Koexpression mit KIR6.2