Acute intracerebral treatment with amyloid-beta (1–42) alters the profile of neuronal oscillations that accompany LTP induction and results in impaired LTP in freely behaving rats
- Sprache
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Englisch
- Identifier
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1205976094
Beteiligte Personen und Organisationen
Kalweit, Alexander Nikolai
Yang, Honghong
Colitti-Klausnitzer, Jens
Fülöp, Livia
Bozsó, Zsolt
Penke, Botond
Manahan-Vaughan, Denise
Yang, Honghong
Colitti-Klausnitzer, Jens
Fülöp, Livia
Bozsó, Zsolt
Penke, Botond
Manahan-Vaughan, Denise
- URN
- Rechteinformation
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
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26.01.2023, 13:55 MEZ
Beteiligte
- Kalweit, Alexander Nikolai
- Yang, Honghong
- Colitti-Klausnitzer, Jens
- Fülöp, Livia
- Bozsó, Zsolt
- Penke, Botond
- Manahan-Vaughan, Denise
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Hippocampal network activity is transiently altered by induction of long-term potentiation in the dentate gyrus of freely behaving rats
Persistent deficits in hippocampal synaptic plasticity accompany losses of hippocampus-dependent memory in a rodent model of psychosis
Synaptic depression in the CA1 region of freely behaving mice is highly dependent on afferent stimulation parameters
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The intriguing contribution of hippocampal long-term depression to spatial learning and long-term memory
Mechanistic flexibility of the retrosplenial cortex enables its contribution to spatial cognition
Learning-facilitated long-term depression and long-term potentiation at mossy fiber : CA3 synapses requires activation of \(\beta\)-adrenergic receptors
Dopamine D1/D5, but not D2/D3, receptor dependency of synaptic plasticity at hippocampal mossy fiber synapses that is enabled by patterned afferent stimulation, or spatial learning
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Hippocampal network activity is transiently altered by induction of long-term potentiation in the dentate gyrus of freely behaving rats
Persistent deficits in hippocampal synaptic plasticity accompany losses of hippocampus-dependent memory in a rodent model of psychosis
Synaptic depression in the CA1 region of freely behaving mice is highly dependent on afferent stimulation parameters
Altered neuronal excitability underlies impaired hippocampal function in an animal model of psychosis
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Absence of Pannexin 1 stabilizes hippocampal excitability after intracerebral treatment with A\(\beta\) (1-42) and prevents LTP deficits in middle-aged mice
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